Tbol dosing

Anecdotal reports from human users suggest 25C-NBOMe to be an active hallucinogen at a dose of as little as 200-500 µg insufflated and 300-600 µg buccaly (with threshold doses even lower), making it only half to a third the potency of LSD . [5] NBOMe-substituted compounds have a diminished absorption rate passing through mucus membranes, but generally remain inactive when taken orally. Buccal , sublingual or insufflated routes of administration are all viable options. Absorption rate buccally and sublingually can be increased when complexed with HPBCD complexing sugar, however the most efficient is nasal administration , which shortens the duration while increasing intensity, but has been attributed to several overdoses due to improper dosing. [6]

Amoxapine possesses a wide array of pharmacological effects. It is a moderate and strong reuptake inhibitor of serotonin and norepinephrine , respectively, [19] and binds to the 5-HT 2A , [26] 5-HT 2B , [27] 5-HT 2C , [26] 5-HT 3 , [28] 5-HT 6 , [21] 5-HT 7 , [21] D 2 , [23] α 1 -adrenergic , [23] D 3 , [24] D 4 , [24] and H 1 receptors [23] with varying but significant affinity , where it acts as an antagonist (or inverse agonist depending on the receptor in question) at all sites. It has weak but negligible affinity for the dopamine transporter and the 5-HT 1A , [28] 5-HT 1B , [28] D 1 , [29] α 2 -adrenergic , [23] H 4 , [30] mACh , [23] and GABA A receptors , [29] and no affinity for the β-adrenergic receptors or the allosteric benzodiazepine site on the GABA A receptor. [29] Amoxapine is also a weak GlyT2 blocker , [31] as well as a weak (K i = μM, EC 50 = μM) δ-opioid receptor partial agonist . [32]

Tbol dosing

t bol dosing

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